The loss of Foxc2 expression in the outflow tract links the interrupted arch in the conditional Foxc2 knockout mouse

Mohammad Khaja Mafij Uddin, Wataru Kimura, Mohammed Badrul Amin, Kasumi Nakamura, Mohammod Johirul Islam, Hiroyuki Yamagishi, Naoyuki Miura

研究成果: Chapter

1 被引用数 (Scopus)

抄録

Congenital heart disease is the most common birth defects, affecting 1 % live births [1]. The cardiovascular system undergoes a series of morphogenetic events to form a heart and an aorta in fetuses. Formation of the heart and aorta requires migration, differentiation, and precise interactions among multiple cells from several embryonic origins [2]. Forkhead box2 (Foxc2) encodes a transcription factor and is expressed in mesodermal tissues, such as the pharyngeal artery, outflow tract endothelial/surrounding mesenchyme, bone, and kidney [3]. Simple knockout of Foxc2 in mouse causes an interrupted aortic arch, ventricular septal defect, cleft palate, and skeletal malformation [4]. The heart is made from primary and secondary heart field progenitors. The primary heart field gives rise to the left ventricle and atria, while the secondary heart field contributes mainly to the right ventricle and outflow tract [5] (Fig. 27.1).

本文言語English
ホスト出版物のタイトルEtiology and Morphogenesis of Congenital Heart Disease
ホスト出版物のサブタイトルFrom Gene Function and Cellular Interaction to Morphology
出版社Springer Japan
ページ211-213
ページ数3
ISBN(電子版)9784431546283
ISBN(印刷版)9784431546276
DOI
出版ステータスPublished - 2016 1月 1

ASJC Scopus subject areas

  • 医学(全般)
  • 生化学、遺伝学、分子生物学(全般)

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